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What is Lipomatous hypertrophy?

Lipomatous hypertrophy of the interatrial septum (LHIS) is a benign cardiac mass characterised as a non-encapsulated mass of fatty tissue that infiltrates the atrial septum first described in 1964.

What causes Lipomatous hypertrophy of the interatrial septum?

Lipomatous hypertrophy of the interatrial septum is a common finding seen on echocardiography with an incidence reported to be 1–8% of the general population. It is more commonly seen in women and in the elderly and is associated with obesity, steroid use, and emphysema.

What is located in the interventricular septum?

The interventricular septum divides the right and left ventricles, running in the plane of the anterior and posterior interventicular grooves.

What is the function of the interventricular septum in the heart?

In addition to separating the ventricles, the interventricular septum also functions as a conduit for part of the conducting system of the heart. The atrioventricular bundle of His, which arises from the atrioventricular node, arborizes within the septum to reach its target points.

What is mild Lipomatous hypertrophy of the atrial septum?

Lipomatous hypertrophy of the atrial septum (LHAS) is an anomaly of the heart. It is characterized by an infiltration of adipocytes into myocytes of the interatrial septum, sparing the fossa ovalis, which gives a characteristic hourglass-shaped image.

What is asymmetric septal hypertrophy?

Asymmetric septal hypertrophy (defined by an echocardiographic interventricular septum to left ventricular free wall thickness ratio of greater than or equal to 1.3 and by the presence of suggestive two-dimensional echocardiographic abnormalities) was found in 28 patients (5%).

What is interventricular septal hypertrophy?

Definition. The dividing wall between left and right sides of the heart, thickens and bulges into the left ventricle. [

How does the interventricular septum form?

Development. The muscular part of the interventricular septum derives from the bulboventricular flange which is developed due to differential growth of primitive ventricle and bulbous cordis. It also gets attached to lower border of spiral septum or the aortico pulmonary septum.

What is the interventricular septum and why is it important?

The interventricular septum separates the left ventricle and the right ventricle. It is muscular at the apex and tapers to a membranous portion at the heart base near the origin of the aorta. Septal defects may occur in any area of the septum, but are most commonly located in the membranous portion.

Is atrial septal aneurysm congenital?

Atrial septal aneurysm (ASA) is a congenital deformity of the interatrial septum with a prevalence of 1-2% in the adult population.

Can lipoma cause heart problems?

Depending on the chamber involved and the size of the mass, they can cause obstruction of flow and congestive heart failure [4, 5]. Embolization is a rare phenomenon because lipomas are typically encapsulated.

Can you live a normal life with HCM?

The good news is that most people have no or minimal symptoms throughout their life and even for those with symptoms, most people with HCM can lead a normal life with treatment and follow-up.

What is lipomatous hypertrophy of the interatrial septum?

Lipomatous hypertrophy of the interatrial septum (LHIS) is a benign disorder characterized by fat accumulation in the interatrial septum (IAS).

How does magnetic resonance imaging show the inferior interventricular septum?

Magnetic resonance imaging showed a large fatty area infiltrating the inferior interventricular septum, nonhomogeneous and hyperintense compared to the myocardial wall on steady-state free precession sequences. The borders were not sharply delineated, and the shape of the infiltrating mass was not rounded.

Is the left ventricular septum normal during exercise?

However, no mitral leak, no obstruction of the left ventricular outflow tract, and no systolic anterior motion were observed, and left ventricular function was normal, even during exercise testing. The ECG showed no increased PR interval and no T-wave anomaly or ventricular arrhythmia ( Figure 1 ).